Huckerbee Ch4
·
No clear association between the site of lesion
and dysphagia presentation.
·
Dysphagia occurs in a high proportion of stroke
patients (30%?)
·
Clinical swallowing examination is therefore the
first step in evaluating all stroke patients.
Patient History
·
Medical
History form patient records – medical charts, : Stroke onset,
characteristics and complications associated with stroke (e.g. intubation).
·
History of dysphagia – do they have this?
·
Patient
interview – rehabilitation setting will warrant a lengthier interview than
acute. Find out if swallowing has changed, if different on different
consistencies. Initially open ended – may ask more closed questions if the
patient has cognitive/comm. Diff.
·
Less than 50% of stroke patients are aware of
dysphagia symptoms – limitations of
interview.
Groher CH 7
Clinical examination of swallowing (CED) = description of
problem, health history, clinical observations and a physical examination.
Warning signs (dysphagia likely):
1.
Confused
mental state – may be difficulties with the eating process since requires
planning + judgement e.g. speaking while eating, going too quickly, large
bolus’.
2.
Dysarthria
(slow, labored, slurred articulation, nasal emission, hoarse/breathy voice) =
due to inherent weakness/ incoordination of muscles common to swallowing.
3.
Excessive
drooling (sialorrhea) – due to motor/sensory impairments of swallowing
mechanism.
4.
Frequent
coughing episodes – sign of potential aspiration – i.e. pooling into the
airway.(protective mechanism to prevent aspiration). – no cough reflex = sign
that most foods may be unsafe unless the patient is dysphraxic + has no
voluntary cough.
5. Weightloss
6.
Complaints
of pain/ obstruction – common complaint of those with neurologic condition
resulting in muscle weakness/ incoordination + those with oesophageal motility
disorders. Cricopharyngeal dysfunction – often point to the thyroid cartilage
level. Pooling in valleculae/ pyriforms – may point to area adjacent to the
larynx. Timing – if early – may be oropharyngeal – if later – oesophageal
problem.
7.
History
of pneumonia – may be associated with neuromuscular incoordination,
weakness of the oropharyngeal swallowing mechanism or esophageal dysfunction.
Clinical observations
·
Mental status –
·
Physical examination:
·
Voice and speech – e.g. ability to sustain a
vowel (quality,pitch, intensity) – Hyper-nasality suggests impaired
palatopharyngeal function.
·
Weight – nutrition?
·
Muscles of facial expression
·
Muscles of mastication – masseter is the largest
(inserts into the mandible), temporalis -
jaw up/ forward/back.
·
Pathologic reflexes e.g. brainstem reflexes –
normally inhibited in adults e.g. the bite reflex – often elicited in those
with severe neurologic lesions by touching the tongue, teeth or lips with a
tongue blade.
·
Oral mucosa – moisture?
·
Dentition
·
Gag reflex – very variable – diminished reflex
is significant in patients with
weakened pharyngeal musculature
·
Tongue – coordinated interaction between the
tongue and the pharyngeal muscles propels the bolus. Look for atrophy,
fasciculations or abnormal reflexes.
·
Sensation – chewing/saliva flow and swallowing =
reflexes depending on sensation.
Swallow summary
1.
Bolus moved into the oropharynx by
lip/tongue/cheek muscles
2.
Swallow reflex initiated
3.
Muscles suspending the larynx contract – larynx
moves up so that the epiglottis can go down into the base of tongue (covering
airway).
4.
Pharyngeal constrictors move the bolus towards
the cricopharyngeal sphincter muscle. Which opens allowing the bolus to travel
to the oesophagus.
Swallowing Assessment
·
Swallowing reflexes are subject to warm up and
fatigue.
·
Aspiration is difficult to evaluate – listen for
air mixing with liquid as a sign of aspiration. Change in face colour, gurgily
breath signs, breathiness or loss of voice may indicate acute aspiration
·
Examine the oral cavity for residue.
·
Ice chips = safe + give sensory stimulus
Logemann chapter 5
Oral –motor control
examination
·
Evaluation of the range rate and accuracy of movements of the lips, tongue, soft
palate and pharyngeal walls during speech, reflexive activity and swallowing.
·
Swallowing apraxia – usually perform best when
no swallowing instructions are given
·
Labial function - /i/ /u/ alternately, drooling/
anterior loss?
·
Tongue – touch mouth corners/ lips + then rapid
tongue movement. Posterior tongue function (tongue base) – get patient to say
/k/ + repeat /ka/ (DDK).
·
Soft palate – ask patient to produce a strong
loud ‘ah’ and to sustain this for a few seconds.
·
Oral sensation - use an oral sponge – ask the patient if everywhere feels
the same
Laryngeal examination
·
Voice quality – gurgily voice = associated with
aspiration, hoarsness – may indicate reduced laryngeal closure – cough reflex
(although voluntary cough doesn’t mean the patient will have a reflexive
cough).
·
Ability to change pitch – pharyngeal swallow +
cough reflex may arise from the superior laryngeal nerve – therefore inability
to change pitch implies reduced sensitivity around the larynx. Phonation time
tasks – info on laryngeal control – e.g. /z/ prolong a /z/ as long as possible.
Phonation time = also a test of respiration.
The Swallowing
Examination
Observations during
swallowing trial
·
Hand under the patients’ chin with fingers
spread and in light contact: index finger behind mandible, middle finger on
hyoid, little finger at the bottom of the thyroid cartilage.
·
Feel for: tongue movement, hyoid movement, and
laryngeal movement. Compare the time between the initiation of tongue movement
and the initiation of hyoid/ laryngeal movement.
·
Can’t assess what is occurring physiologically.
·
Ask patient to say ‘ah’ after swallowing –
listen to vocal quality. (however consider silent aspiration, potentially 50%).
·
Ask patient to pant + turn head to either side
after swallowing – material dislodged from the pharyngeal recesses – and then
say ‘ah’.
·
Use small amounts of material
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